2017 AF Symposium: New Reports on Reversing Fibrosis and Hypercoagulability & NOACs
I’ve been a busy writer since attending the 2017 AF Symposium in January. Here are two more summary reports.
Report 5: Some Forms of Fibrosis May Be Reversible: Research with Overweight Sheep
In a very hopeful study for Atrial Fibrillation patients, Dr. Stanley Nattel of the University of Montreal, Montreal, Canada concluded that some forms or types of fibrosis are indeed reversible.

Steve with Shannon Dickson, editor of The A-Fib Report
He described his experiments with overweight sheep. A 30% weight lost reduced fibrosis as well as inflammation and incidence of A-Fib. Continue reading….
Report 6: Hypercoagulability May Cause A-Fib, NOACs May Prevent It
Novel oral anticoagulants (NOACs) may both prevent and stop A-Fib, according to a thought-provoking hypothesis by Dr. Ulrich Schotten of the University of Maastricht, Maastricht, The Netherlands.
His different, somewhat contrary hypothesis flips the current thinking― that hypercoagulability increases and promotes A-Fib (versus A-Fib increasing hypercoagulability).
Working with specially designed mice with increased thrombin activity (hypercoagulated mice), Dr. Schotten found that these mice had increased atrial fibrosis and A-Fib, and a hypercoagulated state promotes atrial fibrosis…Continue reading….
See the entire list of my reports from the 2017 AF Symposium.
Look for three more reports soon.